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Bell, Graeme
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Item TypeName
Concept Potassium Channels, Inwardly Rectifying
Academic Article Rat inwardly rectifying potassium channel Kir6.2: cloning electrophysiological characterization, and decreased expression in pancreatic islets of male Zucker diabetic fatty rats.
Academic Article Identification of microsatellite markers near the human genes encoding the beta-cell ATP-sensitive K+ channel and linkage studies with NIDDM in Japanese.
Academic Article Linkage studies in NIDDM with markers near the sulphonylurea receptor gene.
Academic Article Treatment of hyperglycemia in a 7-year-old child diagnosed with neonatal diabetes.
Academic Article Permanent neonatal diabetes due to mutations in KCNJ11 encoding Kir6.2: patient characteristics and initial response to sulfonylurea therapy.
Academic Article Insulin gene mutations as a cause of permanent neonatal diabetes.
Academic Article Diagnosis and treatment of neonatal diabetes: a United States experience.
Academic Article Kir6.2 variant E23K increases ATP-sensitive K+ channel activity and is associated with impaired insulin release and enhanced insulin sensitivity in adults with normal glucose tolerance.
Academic Article Neonatal diabetes: an expanding list of genes allows for improved diagnosis and treatment.
Academic Article Association of the KCNJ11 variant E23K with type 2 diabetes in Indo-Trinidadians.
Academic Article Human G-protein-coupled inwardly rectifying potassium channel (GIRK1) gene (KCNJ3): localization to chromosome 2 and identification of a simple tandem repeat polymorphism.
Academic Article G protein-activated inwardly rectifying potassium channel (GIRK1/KGA) mRNA in adult rat heart and brain by in situ hybridization histochemistry.
Academic Article Alternative splicing of human inwardly rectifying K+ channel ROMK1 mRNA.
Academic Article The 31-cM region of chromosome 11 including the obesity gene tubby and ATP-sensitive potassium channel genes, SUR1 and Kir6.2, does not contain a major susceptibility locus for NIDDM in 127 non-Hispanic white affected sibships.
Academic Article 5-amino-imidazole carboxamide riboside acutely potentiates glucose-stimulated insulin secretion from mouse pancreatic islets by KATP channel-dependent and -independent pathways.
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